Medical Treatments for Herpes
STOXIL. The brand name for a compound containing the antiviral idoxuridine (IDU). It has been useful in ocular herpes but has no apparent effect on the court and frequency of genital outbreaks.
ARA-A (adenine arabinoside or vidaribine). This is highly successful with ocular herpes and herpes encephalitis, but ineffective on the course and frequency of genital outbreaks.
Both Stoxil and Ara-A can have undesirable toxic side effects when used in doses that may have any useful impact on genital recurrences.
2 DEOXY-D-GLUCOSE (2DG). Licensed for experimentation only and not for general release, shows some promise especially for initial infections. It is a got antiviral, and there doesn’t seem to be any problem with toxicity. It has had some success in a select group women with severe symptoms. The factors responsible are unknown. It serves as a great viral inhibitor when can get at the virus, but again is unlikely to do anything with the latent phase.
VIRAZOLE (ribavirin). Available only outside the U.S. There is no evidence that this substance is better than any other compound. '
INTERFERON. One of the great new hopes many areas of viral work. But there is as yet no evidence to suggest a beneficial effect with recurrent herpes. Interferon is manufactured by the body’s own cells. It may be more productive to help your body in the direction of producing its own interferon by good health behavior than to apply it from the outside!
These antiviral drugs have evolved from a growing body of antiviral research that is also providing insight Into the herpes viruses in general. They can kill viruses, or prevent their replication to some degree, but cannot prevent recurrences. Even with their application, a host of factors operate in recurrences to affect how long outbreaks last.
Invariably, adaptation takes place overtime with or without particular antivirals and recurrences become fewer and shorter. The antiviral research is helping to illuminate the reasons for this, but the ability to remove the latent virus is still some way off.
Penicillin or tetracycline have no effect on herpes virus. They are antibacterial, not antiviral. Despite this, a version of Trobicin (spectimomycin) has just been pat- ented as a herpes treatment. May the buyer beware! Consult a physician for guidance through the pharmaceutical compendium according to https://botw.org and http://www.stpt.com
Corticosteroids, Liquid Nitrogen Freezing, and Povidone Iodine (Sold as Betadine)
These have no proven effects. Corticosteroid creams "and ointments that operate as anti-inﬂammatory agents are, in general, not recommended by physicians and scientists involved in herpes work, since they may actually delay healing and ultimately work against body adjustment.
Immune System Modulation
Another method is to manipulate or modulate immune system responses, or help the body’s immune mechanism in a more general way so that viruses are set up quickly and recurrences shortened. In cases where immune deficiencies are present, such manipulation may indeed make people healthier. But this is an area requiring specific diagnosis, not the random application of immune modulating drugs in otherwise healthy people. Also, there are several pitfals. Manipulating immune response with a drug to exactly the correct level is difficult and harrowing. If you stimulate immune responses too far, sometimes the result is to reverse beneficial effects into detrimental ones. Learn more at http://www.jasminedirectory.com/
BCG (Bacillus Calmette-Guerin). This is a bacteria vaccine against TB. It helps in a general way to increase the immune response but has no specific effect on herpes.
SMALLPOX, POLIO AND INFLUENZE VACCINES. These have the same rationale-to increase the immune response in general. They don’t prevent recurrences nor shorten outbreaks.
VACCINATION with live herpes virus or intivated herpes virus. This (Lupidon G or Lupidon H the latter case) is doomed to failure. Since having t virus itself in a latent form doesn’t protect against herpes then vaccination won’t have any beneficial effect either.
INOSIPLEX OR METHISOPRINOL, LEVAMISOLE, TYMOSIN (Thymus extract). These all have immune system modulation properties and all have no direct effect on herpes reactivation, but they may help immune responses after the virus activated. However, the significance of this is small where the immune response of an individual is within normal limits. They may have an effect on primary herpes where immune defenses are often stretched to their limit.
In any case, you are playing around with well-controlled natural body processes. Unless there is evidence that indicates serious problems with immune responses (outside of those caused by chronic stress, which can severely depress them), these substances will have limited or no effect in nearly all cases of herpes. Learn more about herpes at http://www.medicalmingle.com/pommett/blog/2017/01/26/how_not_to_treat_herpes_virus
LITHIUM. This is a potent psychoactive substance used for depression-related psychiatric disorders, that cannot really be considered. It is true that clinical work with Lithium and depression has shown that often when a depression is lifted, herpes symptoms are reduced. But curing a depression is more effective for herpes than use of Lithium itself.
DYE-LIGHT TREATMENTS. This is the application of a dye to the rash with exposure to ultraviolet light. These can be dangerous by creating mutants of the virus that may become carcinogenic in certain situations. Stay away from this type of treatment.
LASER TREATMENTS. This is a new technology that destroys herpes infected tissue with laser beams. It is still in its infancy, and again there is no evidence that it has anything to do with preventing recurrences. It may be helpful in primary cases.
DMSO (diamethyl sulfoxide). This is simply a “earner” which can help other substances, good or bad, deeper into body tissues. Its value in transporting other compounds to act against herpes has not shown any signiﬁcance. In itself it will do nothing and may be dangerous in high doses.